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The New England College of Optometry

The New England College of Optometry in Boston, Massachusetts, is the oldest continually operating college of optometry in the United States. It was originally established as the Klein School of Optics in 1894 by Dr. August Andreas Klein, an ophthalmologist. The college moved to several locations around Boston, and was known by a few other names (Massachusetts School of Optometry, Massachusetts College of Optometry) until it came to reside in its current location in the historic Back Bay section of Boston as the New England College of Optometry.

More information: http://en.wikipedia.org/wiki/New_England_College_of_Optometry

Results - con’t References Conclusions Discussion Sample Purpose Methods Results Introduction Results - con’t The Sample Scientific Poster - Using Microsoft Office Power Point Template K. Richard1, M. Jacisin2 1New England Eye Institute, 2New England College of Optometry, Boston, MA

Results - con’t References Conclusions Discussion Sample Purpose Methods Results Introduction Results - con’t The Sample Scientific Poster - Using Microsoft Office Power Point Template K. Richard, W.M.Player New England Eye Institute, Boston, MA

Results - con’t References Conclusions Discussion Sample Purpose Methods Results Introduction Results - con’t The Sample Scientific Poster - Using Microsoft Office Power Point Template K. Richard1, W.M. Player2 1New England College of Optometry, 2New England Eye, Boston, MA

T. Farra1, M. Jacisin2 1Brigham and Womens Hospital, 2New England College of Optometry, Boston, MA Results - con’t References Discussion Sample Purpose Methods Results Introduction Results - con’t The sample was selected from patients aged 40 and older who presented to the Division of Ophthalmology at Brigham and Women’s Hospital from July 1998 to January 1999. Patients were required to have electronic medical records dating back to at least 1996. All patients were examined by a single observer. Each patient was questioned about their past ocular, medical and surgical history, as well as current use of medications. An extensive history of past and present steroid use was obtained which included name of the steroid, dose, frequency of administration, duration, and time period of use. Subsequently, all patients underwent a complete eye examination. Any patient suspected of having glaucomatous optic neuropathy based on the appearance of the optic nerve and nerve fiber layer underwent visual field testing with either the Humphrey or Goldmann perimeters. Patients with unreliable visual fields or no reproducible visual field loss were excluded. Ophthalmic information regarding ocular conditions, surgeries and maximum IOP was corroborated from the patient’s eye records. Non-ophthalmic information was confirmed by a review of the patient’s electronic medical record. These records contained information regarding the patient’s medical conditions, as well as the date ...

The Sample Scientific Poster - Using Microsoft Office Power Point Template T. Farra1, M. Jacisin2 1Brigham and Womens Hospital, 2New England College of Optometry, Boston, MA Results - con’t References Conclusions Discussion Sample Purpose Methods Results Introduction Results - con’t Retinal ganglion cells (RGC) die by apoptosis in both high tension1 and low tension2 glaucoma. Ischemia and neuronal compression may deprive the RGCs of essential trophic factors necessary for their survival3. When apoptosis occurs, the RGC cytoplasmic volume decreases and the cell membrane ruptures, while the nucleus condenses4. RGC membrane lysis is mediated by phospholipases and oxidative enzymes. Leaky RGC may release other toxic substances such as excitatory amino acids, which further damages neighboring cells3. Glutamate, an excitatory amino acid, has been found in elevated concentrations in the vitreous body of glaucomatous eyes5. Steroids are known to be nonspecific blockers of cell membrane lipases6 which may inhibit apoptosis in this setting. Furthermore, certain glucocorticoids are thought to intercalate into the cell membrane and decrease neuronal and vascular membrane fluidity by inhibiting oxygen free radical-induced lipid peroxidation7. Studies have shown that RGC survival in vitro is dependent on the addition of steroids to the culture8. Finally there is data suggesting an immune deviation in patients with NTG2 where serum antibodies directed to retinal elements...
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